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GLP-1 produces therapeutic effects via synchronized activities within the pancreatic, gastrointestinal, and central neurological systems, making it optimal for the dual control of diabetes and obesity (see to Table 1 for comprehensive mechanisms) (Ruze et al

Actin exists in two forms: globular (G-actin) monomers and filamentous (F-actin) polymers

Morphologically, ferroptosis involves mitochondrial shrinkage and increased membrane density, reflecting its unique metabolic imbalances [89, 90]

For those obese patients, doctors may need to prescribe medicines

A dual agonist such as tirzepatide activates both the GLP-1 and the GIP receptor, which the article describes as potentially engaging additional metabolic pathways